Puas yog koj tab tom nrhiav ib qho tshiab uas tuaj yeem txhim kho kev txawj ntse thiab tiv thaiv lub hlwb? Nooglutyl yog ib qho kev sim tshuaj neurotrophic los ntawm Russia uas feem ntau hloov kho glutamate hauv lub hlwb, tshwj xeeb yog AMPA receptors. Nws tau pom tias muaj peev xwm txhim kho kev nco, txhim kho kev kawm, thiab muab kev tiv thaiv neuroprotection, suav nrog kev tiv thaiv hypoxia thiab mob hlwb. Cia peb kawm ntxiv txog nws.
Nooglutyl yog dab tsi
Nooglutyl tshuaj ntsiavyog ib qho kev sim nootropic compound tsim nyob rau hauv Russia, feem ntau kawm nyob rau hauv preclinical thiab tsiaj thwmsim. Nws raug suav hais tias yog glutamate- hloov kho kev paub txog kev txawj ntse, feem ntau ua rau lub paj hlwb excitatory neurotransmitter system koom nrog hauv kev kawm thiab kev nco. Cov kev tshawb fawb qhia tias nws tuaj yeem pab tsim kev nco, txhim kho kev kawm, thiab muab kev tiv thaiv neuroprotection hauv hypoxic lossis mob hlwb qauv. Nws cov txheej txheem ntawm kev ua haujlwm yuav suav nrog kev txhim kho AMPA receptor kev ua haujlwm thiab tswj glutamate signaling, yog li txhim kho synaptic kis tau tus mob thiab tiv thaiv neurons los ntawm overexcitation. Yog tias koj xav tau Nooglutyl, thov hu rau Xi'an Sonwu.
Cov txiaj ntsig ntawm Nooglutyl yog dab tsi
Nooglutyl yog ib qho kev paub txog - txhim kho qhov chaw hauv tebchaws Russia, feem ntau tau kawm txog nws cov teebmeem ntawm kev nco, kev kawm, thiab kev tiv thaiv neuroprotection. Cais raws li ib tug nootropic, nws yog structurally muaj feem xyuam rau noopeptides thiab racetams, tab sis nws lub ntsiab mechanism ntawm kev txiav txim muaj xws li cov kev cai ntawm glutamate thiab AMPA receptors. Cov hauv qab no yog qee yam ntawm nws cov txiaj ntsig tau tshaj tawm lossis tau kawm:
1. Kev Txhim Kho Kev Nco: Kev tshawb fawb tau pom tias nooglutyl txhawb kev tsim ntawm ob qho tib si luv-lub sij hawm thiab ntev- lub sij hawm nco hauv kev sim tsiaj. Nws txhim kho synaptic plasticity- lub hlwb lub peev xwm los tsim kev sib txuas tshiab- ua kom kawm thiab nco cov ntaub ntawv yooj yim dua.
2. Neuroprotection: Nws pab tiv thaiv neurons los ntawm kev puas tsuaj los ntawm hypoxia (tsis muaj oxygen), oxidative kev nyuaj siab, los yog raug mob. Qhov no tuaj yeem muaj txiaj ntsig zoo los tiv thaiv kev poob qis thiab txhawb kev rov qab los tom qab raug mob hlwb.
3. Txhim kho kom pom tseeb thiab kev kawm muaj peev xwm: Los ntawm kev ua kom zoo dua kev sib kis ntawm glutamatergic, nooglutyl tuaj yeem txhim kho kev mloog zoo, kev xav pom tseeb, thiab kev kawm tau zoo.
4. Kev tiv thaiv - ntxhov siab vim thiab lub siab xav- ua kom ruaj khov: Qee cov kev tshawb fawb qhia tias nooglutil tuaj yeem txo kev ntxhov siab-zoo li kev coj cwj pwm thiab pab kom muaj kev sib haum xeeb los ntawm kev tswj cov kev ua haujlwm neurotransmitter hauv hippocampus thiab prefrontal cortex.
5. Txhim kho lub hlwb lub zog metabolism: Nooglutilyl txhawb nqa cov hlab ntsha hauv hlwb kom zoo dua thiab siv cov piam thaj, txhim kho tag nrho lub hlwb ua haujlwm thiab kev puas siab puas ntsws.
6. Muaj peev xwm pab txhawb rau kev puas siab puas ntsws: Nws tau raug tshawb xyuas raws li cov tshuaj sib tw rau kev kho mob amnesia, neurodegenerative kab mob, lossis tom qab- mob stroke kev paub tsis meej.
Txawm hais tias nws qhov kev cia siab zoo, nooglutilyl tseem yog ib qho kev tshawb nrhiav nrog tib neeg cov ntaub ntawv thiab tsis tau txais kev pom zoo siv los ua kev kho mob sab nraud ntawm Russia.
Yuav ua li cas yog lub pharmacological mechanism ntawm Nooglutyl
Lub chaw muag tshuaj ntawm nogrolide tuaj yeem raug suav hais tias yog glutamate -centric neuromodulator uas txhim kho synaptic plasticity thaum muab kev ruaj ntseg thiab kev tiv thaiv rau cov neurons. Txawm li cas los xij, cov txheej txheem no feem ntau yog los ntawm kev tshawb fawb preclinical. Nws cov tshuaj pharmacological feem ntau suav nrog kev tswj hwm ntawm lub hlwb glutamatergic system, uas yog qhov tseem ceeb rau kev kawm, nco, thiab kev sib txuas lus neuronal.
Nws lub luag haujlwm tseem ceeb yog ua tus tswj hwm zoo ntawm AMPA-hom glutamate receptors. Cov receptors no yog lub luag haujlwm rau kev hloov pauv sai ntawm lub hlwb hauv lub hlwb thiab ua lub luag haujlwm tseem ceeb hauv synaptic plasticity (tus txheej txheem tseem ceeb hauv kev tsim kev nco). Qhov sib xyaw no tau xav los txhim kho kev sib kis ntawm synaptic thiab txhim kho neural signal transduction efficiency los ntawm kev nce AMPA receptor kev ua.
Ntxiv rau kev tswj hwm AMPA receptors, qhov sib xyaw no tuaj yeem cuam tshuam ncaj qha rau NMDA receptors, yog li pab tswj cov calcium ion nkag mus rau hauv cov neurons. Qhov no yog qhov tseem ceeb heev vim tias kev ua haujlwm NMDA sib npaug txhawb kev kawm, thaum overactivation ua rau excitotoxicity (neuronal puas). Nws zoo nkaus li pab txhim kho qhov system no, yog li siv nws cov txiaj ntsig neuroprotective.
Lwm qhov ntawm nws cov txheej txheem ntawm kev ua haujlwm suav nrog kev tswj hwm dopaminergic, tshwj xeeb yog me ntsis cuam tshuam rau dopamine D₂ receptors. Qhov no tuaj yeem ua rau muaj kev txhawb zog zoo dua qub, kev coj tus cwj pwm, thiab kev zoo siab nyob hauv kev tshawb fawb tsiaj. Qhov tseem ceeb, qhov sib xyaw ua ke tau pom tias tiv thaiv cov neurons hauv kev ntxhov siab xws li hypoxia lossis ischemia. Cov nyhuv neuroprotective no yuav cuam tshuam rau nws cov kev cai ntawm glutamate signaling, yog li txo qis overexcitation thiab tswj cellular kev ncaj ncees thaum lub sij hawm metabolic stress.
Qhov Kev Kawm Dose Nooglutyl yog dab tsi
(Kev Tshawb Fawb Cov Ntaub Ntawv Ceeb Toom: Cov ntaub ntawv noj tshuaj hauv qab no yog ua raws li kev tshawb fawb txog kev kho mob thiab cov neeg siv cov lus ceeb toom, tsis yog cov lus qhia kho mob.)
1. Kev Kawm Txuj Ci
Kev tshawb fawb tsiaj: Sib npaug rau 10-30 mg ib kg lub cev hnyav (kwv yees li 10-40 mg ib hnub rau tib neeg). Kev siv tib neeg (qhia hauv kev tshawb fawb Lavxias thiab Nooglutylene zej zog):
Feem ntau: 10-40 mg txhua hnub
Thawj koob tshuaj: 10-20 mg ib hnub ib zaug
Cov koob tshuaj ntau tshaj plaws: Txog 60-80 mg txhua hnub, muab faib ua ob zaug (thaum sawv ntxov thiab yav tav su).
2. Txoj Kev Tswjhwm
Feem ntau siv rau qhov ncauj, tab sis siv rau qee cov tsiaj hauv kev tshawb fawb thaum ntxov.
Nws raug nquahu kom noj nws ib zaug txhua hnub thaum sawv ntxov vim nws tuaj yeem ua rau muaj kev ceeb toom me ntsis. tuaj yeem noj nrog zaub mov lossis ntawm lub plab khoob (qhov sib txawv ntawm qhov nqus yog tsawg).
3. Pib thiab Lub Sijhawm Ua Haujlwm
Lub sij hawm pib: Tsis pub dhau 30 mus rau 60 feeb
Duration: 6-10 teev (qee cov neeg siv qhia ntau dua, cov teebmeem tsis zoo ntawm kev xav thiab kev xav). Kev siv lub voj voog: Ntau tus neeg siv ua raws li kev tswj hwm ntawm 4-8 lub lis piam, tom qab ntawd so 2-4 lub lis piam txhawm rau tiv thaiv kev zam.
Yog xav paub ntxiv txog tus nqi ntawm nooglutyl capsule lossis cov ntaub ntawv khoom ntxiv, thov hu ncaj qha rau Xi'an Sonwu.
Email:sales@sonwu.com